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In HBECs, the increase in ROS levels triggered by CSE exposure was likely a key factor leading to the translocation of Drp1 to the mitochondria and subsequent induction of mitochondrial fission, and effective inhibition of Drp1 translocation and mitochondrial fragmentation by the ROS scavenger N-acetylcysteine (NAC) supported this view ( 5.3 Mitochondrial biogenesis and COPD Mitochondrial biogenesis is crucial for maintaining cellular homeostasis and it enables cells to replace damaged or aged mitochondria with new ones, thus maintaining normal morphology, structure, and function of mitochondria

If the other tenant is violating those rules, it provides a clear basis for addressing the problem