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When oxidative damage accumulates again to a certain level, ferroptosis is also induced in these originally adjacent cells, which continues to lead to a reduction in the antioxidant capacity of surrounding cells, ultimately leading to large areas of pRTECs damage
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[2] In all these tissues, a constant supply of NADPH is required to sustain reductive biosynthesis and/or to counteract reactive oxygen species (ROS), which can damage sensitive cellular structures such as DNA, membrane lipids, and proteins
This indicates that metabolite profiling of the gut-renal-cerebral axis reveals a particular pattern in different stages of kidney diseases
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